By Ad Rijnberk, Hans S. Kooistra
This hugely illustrated textbook is geared toward the veterinarian in perform or education drawn to every kind of endocrine illnesses of canine and cats. The e-book is organised via physique method and is case oriented.
Chapters take care of separate endocrine glands, starting with a short evaluation of the morphology and body structure of the gland, whole with drawings depicting the practical rules. this can be via descriptions of the issues of the gland with up to date info on analysis and treatment.
Numerous medical instances illustrate the proposing signs and the implications following therapy. the ultimate chapters include diagnostic and healing protocols, in addition to algorithms for difficulties similar to alopecia, polyuria, and weightloss.
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Extra resources for Clinical Endocrinology of Dogs and Cats: An Illustrated Text, Second, Revised and Extended Edition
112,113 The most frequently reported treatment for feline acromegaly has been radiation therapy. 2. 1, the release of GH from mammary tissue is a normal physiological process in dogs during the luteal phase of the estrous cycle. In some middle-aged and older bitches sufficient amounts of GH may be released to result in acromegaly (and diabetes mellitus). 123,124 Progestins also induce GH expression in mammary tissue in cats125, but the GH does not reach the systemic circulation126 and consequently does not lead to acromegaly.
Normochromic normocytic anemia has been found in dogs treated with pharmacological doses of porcine GH, and is associated with depletion of the erythroid cell series as well as cellular atrophy in the bone marrow. 100 Now that more than 100 cases of acromegaly have been described in cats, it is a well-recognized syndrome. It is primarily a disease of castrated males, six to 15 years of age. In principle the physical changes are the same as in the dog but usually less pronounced (fig. 20). Almost all of the affected cats are presented because of poorly controllable diabetes mellitus, due to GH-induced insulin resistance.
The owner had noticed that it was becoming larger, with a heavy head. 2 mm in width (arrow). Also visible are thick mucosal folds of the palatum molle which almost completely obliterated the nasopharynx (arrowhead). Three weeks after transsphenoidal hypophysectomy, the cat no longer required insulin. 97 The soft tissue overgrowth included thickening of the skin, particularly of the head and neck, and enlargement of the tongue with respiratory stridor. The osseous changes caused widening of the interdental spaces, increasing stiffness, difficulty in standing up, and neck rigidity – due to articular cartilage proliferation, periarticular periosteal reaction, and severe spondylosis deformans.